Lesions of the dentin and their significance in the production of dental caries

by E. Wilfred Fish

Publisher: s.n. in [S.l

Written in English
Published: Pages: 1008 Downloads: 836
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Subjects:

  • Dentin.,
  • Dental caries.

Edition Notes

Other titlesJournal of the American Dental Association.
Statementby E. W. Fish.
The Physical Object
Paginationp. [992]-1008 :
Number of Pages1008
ID Numbers
Open LibraryOL18598064M

  The aim of this systematic review and network meta-analysis was to verify the effect of the treatments on caries lesion arrestment (CLA) or the success rate (SR) of dentin caries lesion treatments in the primary teeth. Methods A search was conducted using the MEDLINE/PubMed, Web of Science and Scopus databases through December Proteolysis in the initiation of dental caries is likely to be of no significance, but its role in the progression of the more advanced carious lesions cannot be ruled out. 5. The proteolysis-chelation theory. Simultaneous microbial degradation of the organic components and the dissolution of the minerals of the tooth by the process known as.   Introduction. Classically dental caries is defined as a multifactorial disease whose development requires a susceptible host, cariogenic bacteria, and a contribution from the diet; 1 even if nowadays a new paradigm of dental caries has been postulated. This latter asserts that the caries process is the ubiquitous, natural metabolism in the biofilm that causes fluctuations in pH. Understanding Dental Caries: From Pathogenesis to Prevention and Therapy Michel Goldberg (eds.) This book thoroughly explains the biological background of dental caries and the formation of carious lesions, providing the reader with a sound basis for understanding the role and effectiveness of different therapeutic and preventive measures.

Figure Caries in enamel versus dentin. Image credit “Tooth “By ADuran is licensed CC BY-SA Dentin caries. Once a dental caries spreads through enamel, when it reaches the DEJ it spreads at an increased rate. The triangle pattern of enamel loss is quickly repeated in a new triangle of a dentin caries. It is possible, but not. The hypothesis is that a restoration with an adequate peripheral seal, placed over a cavitated dentin lesion, can arrest the progress of caries lesions. The sealing of the cavity contributes to the formation of tertiary dentin and sclerosis of dentinal tubules, thus preventing pulp exposure [40]. During preparation, complete caries excavation based on clinical judgment (i.e., the color and texture of dentin in the cavity preparation) does not provide certainty as to whether bacteria remain. Caries bacteria present in dentinal tubules subjacent to deep dentinal lesions can be recovered from the nonexposed pulp tissue in the majority of. Recently, with the development of better risk assessment tools and newer technologies that can identify the presence of dental caries lesions at an earlier stage with improved specificity, medicinal therapeutic interventions have been introduced, including the use of silver diamine fluoride products along with techniques such as resin.

  In dental caries, oral bacteria generates acids that diffuse into enamel and further into dentin, the most abundant mineralized tissue in the tooth, dissolving mineral, and eventually destroying the matrix. As a result of this process the mechanical properties of .   “Dental caries is determined by the dynamic balance between the pathological factors that lead to demineralization and the protective factors that lead to remineralization.” [] Caries is a tissue consisting of densely packed crystallites formed in a single axis having both inter- and intra-prismatic micropores measuring between 1 and 30 nm in width.   Dental caries, also known as tooth decay or a cavity, is an infection, usually bacterial in origin, that causes demineralization of the hard tissues (enamel, dentin, and cementum) and destruction of the organic matter of the tooth, usually by production of acid by hydrolysis of the food debris accumulated on the tooth surface. Dental caries is a biofilm-sugar dependent disease 1 Fejerskov O. Changing paradigms in concepts on dental caries: consequences for oral health care. Caries Res ;38(3) that leads to a progressive dissolution of mineralized dental tissues (enamel, dentin, and cementum) until a carious lesion is clinically visible. Despite a worldwide decline in dental caries largely due to the.

Lesions of the dentin and their significance in the production of dental caries by E. Wilfred Fish Download PDF EPUB FB2

Reactions of the Dentin to Advancing Caries* * From the Research Laboratory of the School of Dentistry, University of Louisville. Read before the Section on Histology, Physiology, Pathology, Bacteriology and Chemistry (Research) at the Seventy-Fourth Annual Session of the American Dental Association, Buffalo, N.

Y., Sept. 14, Cited by: 4. The histopathology of caries varies in pre-cavitated and post-cavitated lesions Enamel caries tends to occur on smooth surfaces or pits/fissures Pre-cavitated enamel lesions are composed of 4 zones – translucent zone, dark zone, body of lesion and surface zone.

When a dentist chooses to enter a tooth and remove the caries, he or she can classify the lesion in dentin as rapidly progressing or slowly progressing, according to the guidelines outlined by Fontana and colleagues.

7 This categorization is an oversimplification, as noted by many of the same authors: the carious lesion in enamel can, between 2 time points: (a) progress (exhibit net mineral Cited by: 7. Dental caries is increasing among the elderly (Chapter 19). This phenomenon may be attributed to the fact that more adults are retaining their teeth through the life span.

Studies show that the elderly are experiencing dental caries at a greater rate than children. 49 Certain risk factors are associated with aspects of aging, including xerostomia. Introduction Dental caries is a highly prevalent disease that is considered a major public health problem.

Signs of the caries process extent from the first molecular changes in the apatite crystals of the tooth structure to a white-spot lesion, through dentin involvement and eventual cavitations [ 1 ].Author: Abeer ElEmbaby.

CHRONIC CARIES These lesions are usually of long-standing involvement, affect a fewer number of teeth, and are smaller than acute caries.  Pain is not a common feature because of protection afforded to the pulp by secondary dentin  The decalcified dentin is dark brown and leathery.

 Pulp prognosis is hopeful in that the deepest of lesions usually requires only prophylactic. Essentials of Dental Caries The Disease and Its Management, 3rd Ed (Kidd, Essentials of Dental Caries) Thao Dang Thi Nhu.

Download PDF Download Full PDF Package. This paper. A short summary of this paper. 37 Full PDFs related to this paper.

CARIES OF DENTIN:  Begins with the natural spread of the process along the Dentino-enamel junction and rapid involvement of great number of dentinal tubules.  Act as a tract leading to the dental pulp along which the microorganisms may travel at a variable rate of speed.

SHAFERS TEXT BOOK OF ORAL PATHOLOGY -(extends into inner 1/3 of dentin, possibly invading pulp chamber)-Full cavitation through the enamel-The dentin is clinically exposed-In epidemiologic studies, these lesions are classified as "decayed" (make sure to x-ray before probing, the tooth can crumble and will have no documentation of carious lesion).

Diagnosis consists of both identifying caries lesions and managing them, and it is based on the logic that caries can be prevented, slowed or stopped if it is detected and treated before it causes serious symptoms that require invasive intervention.

author book Dental caries: the disease and its clinical management, the page 3rd edition of which was published in This shorter page text Essentials. A caries management protocol is a comprehensive organized plan designed to arrest or remineralize early caries lesions, eradicate overt caries lesions, and prevent the formation of new lesions in an individual who has a moderate or high rate of caries activity or is at increased risk for developing new caries lesions in the future.

A New Concept of the Caries Problem and Its Clinical Application. Part II Bernhard Gottlieb, Dallas, Texas Dallas Texas Bibliography 1. E.C. Hinds, J. Res. (October ) 2. This will not only save you from the pain of undergoing dental procedures but also save your time and money. In non-cavitated lesions where the infection has not spread to the dentin, remineralization using fluoride toothpaste can reverse the process.

Specific treatment modalities can quickly help to stop the process of caries. Although it is difficult to mimic the features of natural caries lesions, several protocols of artificial carious lesion induction in dentin have been described in in vitro studies.

4,19,20, Cavitated lesion, especially if dentin is involved, pathogenicity of Lactobacillus in dental caries and their ecology in the oral cavity.

View. Statistical significance was accepted as p. under the occlusal surface for changes in the dentin Dental caries is a pathological process consisting of localized destruction of dental soft tissues.

The proximal surface caries grading system suggested by Haugejorden and Slack has 10 categories of carious lesions. T or F. False, There are 4 classes of dental caries recommended by. Micro-Organisms and Caries* * Read before the Peoria County Dental Society, Dec.

2, From the Research Laboratory of the School of Dentistry, University of Louisville. Work supported in part by a grant from the Research Commission of the American Dental Association. Theodore B. Beust, D.D.S. Louisville, Ky. Louisville Ky 1. Fig. Extensive active caries in a young adult (same patient as in Fig.

A, Mirror view of teeth No. B, Cavitated lesions (a) are surrounded by extensive areas of chalky, opaque demineralized areas (b).The presence of smooth-surface lesions such as these is associated with rampant caries.

Occlusal and interproximal smooth-surface caries usually occur in advance of facial smooth. Caries Diagnosis for Smooth Surfaces The following indicate that the lesion is arrested: Caries Diagnosis (contd.) Cavitated lesion, often dark brown, with hard dentin at their bases, the lesions are not plaque covered and are often remote from gingival margin.

Shiny white or brown lesion, often well exposed due to recession, the lesion are not. SEM on ex vivo teeth showed areas of hypermineralization in the intertubular dentin and few blocked tubules, while EDS detected Ag in the center of the lesion.

Enamel-dentin caries, also known as hidden caries, is a phenomenon that leads to formation of highly mineralized, strengthened enamel surfaces, under which the loss of mineral may progress gradually and the carious lesion might extend into dentin without a clinically visible crack at the enamel surface.

There is no consensus on the cause of. Tooth decay, also known as dental caries or cavities, is the breakdown of teeth due to acids made by bacteria. The cavities may be a number of different colors from yellow to black.

Symptoms may include pain and difficulty with eating. Complications may include inflammation of the tissue around the tooth, tooth loss, and infection or abscess formation. Deep caries lesions are cavitated caries lesions that radiographically extend more than 70 to 75 percent into dentin. When the traditional caries removal technique is used to treat the deep caries lesions of vital asymptomatic teeth, the risk of pulp exposure is high.

effect of a variety of bacterial species in dental caries, the chief initiator of caries on tooth enamel is Streptococcus mutans Other species, such as Lactobacilli, have been associated with advancing dental caries lesions, as well as Actinomyces on root cementum.9,12 These various species reside in biofilm on tooth surfaces – dental.

Several studies used dentin modified substrate for different methodologies to produce dentin caries-like lesions in vitro, in an attempt to simulate the caries-affected or carious dentin, and they are used in bond strength tests, remineralization studies and secondary caries inducing in restored teeth (5, 7, 8).

Dental caries Greatest degree of sensitivity experienced when dental caries passes the dental- enamel junction.

As caries As caries penetrates. The lesion may be asymptomatic if the infection is chronic and a path of drainage is achieved. Etiology: This lesion is caused by a dental-related abscess, which has perforated alveolar bone and formed a path of drainage. Treatment: Elimination of the focus of infection through incisional drainage, root canal therapy, or tooth extraction.

Pain. Cervical dentin hypersensitivity (CDH) and noncarious cervical lesions (NCCLs) are common findings in modern clinical practice. Although research has shown that NCCLs are a multifactorial condition involving the three mechanisms of stress, biocorrosion, and friction.

Interpretation of Dental Caries. lesion on the distal surface of the mandibular second premolar. Try to locate the carious lesion.

Classify. A moderate carious lesion on the distal surface of the mandibular second molar. Try to locate the carious lesion. Classify. An advanced carious lesion that extends through the DEJ and into the dentin.

Start studying Caries progression and dentin. Learn vocabulary, terms, and more with flashcards, games, and other study tools. (color depending on chronicity of lesion, due to trapped organic debris) there is a clear demarcation b/w infected and affected dentin in what type of dental caries.

slowly advancing lesions. YOU MIGHT ALSO LIKE.The earliest visual clinical sign of dental caries is the “white spot lesion.” When this is first seen, the carious process has been going on for months.

Figure 2 shows a cross-section of a white spot lesion. Even though the surface appears intact the lesion is at least microns in depth.Current challenging problems in the field are analyzed and the latest research findings, presented.

After an introductory chapter on tooth development, the relationships of biofilm and saliva to dental caries and the significance of the balance between demineralization and remineralization for the development of carious lesions are discussed.